Page 151 - Microsoft Word -Cow Book 10-06-09 final update.doc

E. Estrogens

 Estradiol

F. Testosterone

IV. Non-Steroidal Anti-Inflammatory Drugs (NSAIDs)

These drugs inhibit the conversion of arachidonic acid to prostaglandins
and thromboxanes. This inhibition results in decreased formation of some, but
by no means all mediators of inflammation. These drugs also have some
analgesic, antipyretic effects.

 Aspirin  Meclofenamic Acid
 Phenylbutazone  Naproxen
 Flunixin  Ketoprofen
 Dipyrone  Carprofen

A. Antihistamines

Antihistamines are administered to counter some of the signs associated with
allergic and anaphylactic reactions. Empirically, the antihistaminic agents
seem to be of value in the treatment of various forms of dermatitis , laminitis,
asthma, bloat, mastitis, and metritis. However, there are no well-controlled
clinical studies to support these proposed indications

 Pyrilamine
 Tripelenamine
 Diphenhydramine

B. Diuretics

Diuretics, except water and osmotic diuretics, exert their action by interfering
with the normal kidney transport mechanisms which principally move sodium
back into the system from the provisional urine. The sodium draws additional
water into the urine and a diuresis results. Many edematous conditions such
as udder edema, are associated with a positive-sodium balance. Thus use of
diuretics that interfere with sodium transport mechanisms are the most
effective in the treatment of this class of edema.

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